ABSTRACT
La pancreatitis aguda o crónica puede resultar en lesiones del conducto pancreático, además, la corrosión por el jugo pancreático puede provocar la ruptura de la pared vascular periférica, lo que conduce a hemosuccus pancreático (HP) definido como el sangrado del conducto pancreático a través de la ampolla de Vater. El sangrado suele ser intermitente, repetitivo y, a menudo, no lo suficientemente grave, como para causar inestabilidad hemodinámica. La mayoría de los pacientes tiene antecedentes de enfermedades pancreáticas originales y cuando esto no se cumple, debe incluirse en el diagnóstico diferencial para alcohólicos crónicos con hemorragia digestiva alta intermitente. Presentamos una forma clínica atípica en un paciente masculino de 55 años de edad, con antecedentes de HTA, alcoholismo y aneurisma de la aorta abdominal, quien consultó por dolor tipo cólico en abdomen superior, náuseas y vómitos; luego se asoció disminución del estado de conciencia, alternando con episodios de agitación psicomotriz y primo convulsión generalizada. Una vez ingresado, sucedieron varios episodios de melena. La tomografía abdominal mostró aumento de tamaño y densidad (unidades hounsfield de 58-61) en cabeza y cuerpo del páncreas, por lo que se realizó una angiografía abdominal con protocolo de páncreas y se evidenció doble lesión aneurismática de la arteria esplénica y la arteria gástrica. Falleció en el postoperatorio(AU)
Acute or chronic pancreatitis can result in lesions of the pancreatic duct; in addition, corrosion by the pancreatic juice can cause the rupture of the peripheral vascular wall, which leads to pancreatic hemosuccus defined as bleeding from the pancreatic duct through the Vater ampulla. Bleeding is often intermittent, repetitive, and often not severe enough to cause hemodynamic instability. Most of the cases have a history of strictly pancreatic original diseases, when this is not the case, the PH should be included in the differential diagnosis for chronic alcoholics with intermittent upper gastrointestinal bleeding, We report here an atypical presentation in a 55-year-old male patient with a history of hypertension, alcoholism and an abdominal aortic aneurysm. He consulted for colicky abdominal pain in the upper abdomen, nausea and vomiting; subsequently he presented decreased consciousness, alternating with episodes of psychomotor agitation and generalized seizures. Once hospitalized he had several bouts of melena. The abdominal tomography showed an increase in the size and density (hounsfield units of 58-61) of the head and body of the pancreas. An abdominal angiography with a pancreas protocol was performed, which evidenced a double aneurismal lesion of the splenic and gastric arteries. The patient died after surgery(AU)
Subject(s)
Humans , Male , Middle Aged , Pancreatic Ducts/physiopathology , Pancreatitis/pathology , Alcoholism/complications , Hemorrhage , Pancreatic Juice , Endoscopy , Vascular System Injuries , Internal MedicineSubject(s)
Humans , Triglycerides/analysis , Gallstones/surgery , Gallstones/complications , Pancreatitis, Acute Necrotizing/diagnosis , Pancreatitis, Acute Necrotizing/etiology , Pancreatitis, Acute Necrotizing/pathology , Alcoholism/complications , Pancreatitis, Chronic/diagnosis , Pancreatitis, Chronic/etiology , Pancreatitis, Chronic/pathology , Pancreatic Ducts/physiopathology , Fibrosis/diagnosis , Hemorrhage/diagnosis , NecrosisSubject(s)
Humans , Male , Adult , Pancreas/physiopathology , Pancreatic Ducts/physiopathology , Necrosis/diagnostic imaging , Pancreas/diagnostic imaging , Pancreatic Ducts/diagnostic imaging , Cholangiopancreatography, Endoscopic Retrograde , Pancreatitis, Acute Necrotizing/complications , Cholangiopancreatography, Magnetic ResonanceABSTRACT
Pancreatic duct stones are a common complication during the natural course of chronic pancreatitis and often contribute to additional pain and pancreatitis. Abdominal pain, one of the major symptoms of chronic pancreatitis, is believed to be caused in part by obstruction of the pancreatic duct system (by stones or strictures) resulting in increasing intraductal pressure and parenchymal ischemia. Pancreatic stones can be managed by surgery, endoscopy, or extracorporeal shock wave lithotripsy. In this review, updated management of pancreatic duct stones is discussed.
Subject(s)
Humans , Abdominal Pain/etiology , Catheterization , Calcinosis/complications , Calculi/diagnosis , Endoscopy/instrumentation , Evidence-Based Medicine , Lithotripsy , Pancreatic Ducts/physiopathology , Pancreatitis, Chronic/complications , Sphincterotomy, Endoscopic , Stents , Treatment OutcomeABSTRACT
No abstract available.
Subject(s)
Adult , Female , Humans , Cholangiopancreatography, Endoscopic Retrograde , Manometry , Pancreatic Ducts/physiopathology , Sphincter of Oddi Dysfunction/diagnosis , Tomography, X-Ray ComputedABSTRACT
We report a 6 years old girl with pancreatitis, presenting as recurring bouts of abdominal pain and hyperamylasemia, secondary to a lack of communication between the main papilla and pancreatic duct (pancreas divisium). The diagnosis was made during an intraoperative pancreatography and treatment consisted in a sphincteroplasty of the secondary papilla
Subject(s)
Humans , Female , Pancreatitis/etiology , Pancreatic Ducts/physiopathology , Chronic Disease , Cholangiopancreatography, Endoscopic RetrogradeABSTRACT
Twenty three children with recurrent episodes of diarrhea and chronic malnutrition were studied for pancreatic duct function. Those children were subjected to pancreatic stimulation with pancreozymin and secretin. Grade I malnourished children, as per Gomez classification, formed the control group. The water output from pancreas increased in malnourished children (p < 0.05). It correlated significantly to cationic transport (p < 0.01). Sodium and potassium together accounted for significant proportion of water output in pancreatic fluid. Potassium transport increased with increasing severity of malnutrition and may be responsible for the hypokalemia observed in malnourished children. Pancreatic secretion of bicarbonate decreased in severe malnutrition inspite of increased flow rate of pancreatic secretion. This is probably due to defective bicarbonate secretion likely to be located at pancreatic duct epithelial cell membrane.
Subject(s)
Bicarbonates/metabolism , Case-Control Studies , Child , Child, Preschool , Chronic Disease , Diarrhea/complications , Humans , India , Infant , Ion Transport , Pancreatic Ducts/physiopathology , Protein-Energy Malnutrition/complications , Regression Analysis , Trypsin/metabolismABSTRACT
La fístula pancreática, complicación poco frecuente en el tratamiento de la patología pancreática como extrapancreática, puede curar con tratamiento médico conservador en un 70 por ciento y 80 por ciento de los casos. Cuando éste fracasa, es necesario recurrir al tratamiento quirúrgico, pudiendo ser de tipo resectivo o derivativo de acuerdo a la localización de la fístula. Presentamos un caso de fístula pancreática crónica de etiología traumática, en quien fracasó todo intento de tratamiento médico conservador, siendo necesario la cirugía. Se hace una revisión general del tema